Journal article

Mechanisms of Stress (Takotsubo) Cardiomyopathy


Authors listSzardien, Sebastian; Moellmann, Helge; Willmer, Matthias; Akashi, Yoshihiro J.; Hamm, Christian W.; Nef, Holger M.

Publication year2013

Pages197-19+

JournalHeart Failure Clinics

Volume number9

Issue number2

ISSN1551-7136

DOI Linkhttps://doi.org/10.1016/j.hfc.2012.12.012

PublisherElsevier


Abstract
Mechanisms of Stress (Takotsubo) Cardiomyopathy Sebastian Szardien, Helge Mollmann, Matthias Willmer, Yoshihiro J. Akashi, Christian W. Hamm, and Holger M. Nef Stress cardiomyopathy is a form of reversible systolic dysfunction of the mid and apical left ventricle with pathologic changes of the electrocardiogram in the absence of an obstructive coronary artery disease. The prevalence of stress cardiomyopathy among patients with symptoms. suggestive of myocardial infarction is 0.7% to 2.5%, and it is found predominantly in postmenopausal women (90%). No large studies have confirmed the cause of stress cardiomyopathy. Published data suggest that substantially elevated plasma catecholamine levels, due to emotional or physical stress, may be relevant.



Citation Styles

Harvard Citation styleSzardien, S., Moellmann, H., Willmer, M., Akashi, Y., Hamm, C. and Nef, H. (2013) Mechanisms of Stress (Takotsubo) Cardiomyopathy, Heart Failure Clinics, 9(2), pp. 197-19+. https://doi.org/10.1016/j.hfc.2012.12.012

APA Citation styleSzardien, S., Moellmann, H., Willmer, M., Akashi, Y., Hamm, C., & Nef, H. (2013). Mechanisms of Stress (Takotsubo) Cardiomyopathy. Heart Failure Clinics. 9(2), 197-19+. https://doi.org/10.1016/j.hfc.2012.12.012



Keywords


ANIMAL-MODELAPICAL BALLOONING SYNDROMECATECHOLAMINESCLINICAL-FEATURESContractile dysfunctionLEFT-VENTRICULAR DYSFUNCTIONmitral regurgitationStress cardiomyopathyST-SEGMENT ELEVATIONTakotsubo cardiomyopathyTAKO-TSUBO CARDIOMYOPATHY

Last updated on 2025-02-04 at 02:31