Journalartikel

Cholinergic mechanisms in the neurocontrol of the branchial heart of the cephalopod Sepia officinalis L.


AutorenlisteGebauer, M; Versen, B

Jahr der Veröffentlichung1998

Seiten13-20

ZeitschriftComparative Biochemistry and Physiology. Part C: Pharmacology, toxicology and endocrinology

Bandnummer119

Heftnummer1

ISSN1532-0456

eISSN1878-1659

DOI Linkhttps://doi.org/10.1016/S0742-8413(97)00162-X

VerlagElsevier


Abstract
Cholinergic mechanisms controlling the branchial heart of the cephalopod Sepia officinalis L. were examined. In vitro tests indicate that acetylcholine (ACh) exerts a dose dependent negative inotropic and chronotropic effect. Although the nicotinic agonists carbachol and nicotine mimic the effects of ACh closely, the muscarinic agonists pilocarpine, muscarine and oxotremorine induce only a slight inhibition of amplitude and frequency. Other agents tested (including atropine, pirencepine, hexamethonium, tetraethylammonium (TEA), d-tubocurarine and alpha-bungarotoxin) confirm chat the acetylcholine receptor in branchial heart muscle is probably nicotinic. (C) 1998 Elsevier Science Inc.



Zitierstile

Harvard-ZitierstilGebauer, M. and Versen, B. (1998) Cholinergic mechanisms in the neurocontrol of the branchial heart of the cephalopod Sepia officinalis L., Comparative Biochemistry and Physiology. Part C: Pharmacology, toxicology and endocrinology, 119(1), pp. 13-20. https://doi.org/10.1016/S0742-8413(97)00162-X

APA-ZitierstilGebauer, M., & Versen, B. (1998). Cholinergic mechanisms in the neurocontrol of the branchial heart of the cephalopod Sepia officinalis L.. Comparative Biochemistry and Physiology. Part C: Pharmacology, toxicology and endocrinology. 119(1), 13-20. https://doi.org/10.1016/S0742-8413(97)00162-X



Schlagwörter


AGONISTSANTAGONISTSAPLYSIA NEURONSbranchial heartbungarotoxincephalopodinvertebratemolluscMONOAMINESnicotinic receptorpharmacologySepia

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